Log on / register
BioMed Central home | Journals A-Z | Feedback | Support | My details
Multiple pathways contribute to the pathogenesis of Huntington disease.
Li S, Li XJ
Mol Neurodegener 2006, 1:19
[Full text] [PubMed] [Related articles] [Cited on BioMed Central]


printer friendly 
to  [Help]

PubMed Central articles that cite the above article:

1.
Impaired ubiquitin-proteasome system activity in the synapses of Huntington's disease mice.
Wang J, Wang CE, Orr A, Tydlacka S, Li SH, Li XJ
J Cell Biol 2008 Mar 24, 180:1177-89
 [PubMed Central] [PubMed] [Related articles]
2.
Intracellular degradation of misfolded proteins in polyglutamine neurodegenerative diseases.
Li X, Li H, Li XJ
Brain Res Rev 2008 Nov, 59:245-52
 [PubMed Central] [PubMed] [Related articles]
3.
N-terminal mutant huntingtin associates with mitochondria and impairs mitochondrial trafficking.
Orr AL, Li S, Wang CE, Li H, Wang J, Rong J, Xu X, Mastroberardino PG, Greenamyre JT, Li XJ
J Neurosci 2008 Mar 12, 28:2783-92
 [PubMed Central] [PubMed] [Related articles]
4.
Differential activities of the ubiquitin-proteasome system in neurons versus glia may account for the preferential accumulation of misfolded proteins in neurons.
Tydlacka S, Wang CE, Wang X, Li S, Li XJ
J Neurosci 2008 Dec 3, 28:13285-95
 [PubMed Central] [PubMed] [Related articles]
5.
Huntingtin interacts with the cue domain of gp78 and inhibits gp78 binding to ubiquitin and p97/VCP.
Yang H, Liu C, Zhong Y, Luo S, Monteiro MJ, Fang S
PLoS One 2010, 5:e8905
 [PubMed Central] [PubMed] [Related articles]

This article is also cited by 3 articles in BioMed Central

printer friendly 
to  [Help]
Terms and Conditions Privacy statement Information for advertisers Jobs at BMC Contact us
© 1999-2010 BioMed Central Ltd unless otherwise stated. Part of Springer Science+Business Media.