Autoimmune-induced preferential depletion of myelin-associated glycoprotein (MAG) is genetically regulated in relapsing EAE (B6 × SJL) F1 mice

doi:10.1186/1750-1326-3-7

Molecular Neurodegeneration

Volume 3

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Research article

Autoimmune-induced preferential depletion of myelin-associated glycoprotein (MAG) is genetically regulated in relapsing EAE (B6 × SJL) F1 mice

Dusanka S Skundric¹^,2 , Rujuan Dai¹ , Vaagn L Zakarian³ and Weili Zhou¹

¹Department of Neurology Wayne State University School of Medicine, Detroit, USA

²Department of Immunology and Microbiology, Wayne State University School of Medicine, Detroit, USA

³Department of Computer Science, University of Colorado, Denver, USA

author email corresponding author email

Molecular Neurodegeneration 2008, 3:7doi:10.1186/1750-1326-3-7


Published:	9 June 2008

Additional files

Additional file 1:

Two-color MBP and NF160 specific immunostaining in spinal cord of relapsing H-2^b/smouse. Lack of co-localization between MBP and NF160 immunostaining may be found, as well as areas of co-localized immunostaining.

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Additional file 2:

Two-color MBP and NF160 specific immunostaining in spinal cord of relapsing H-2^bmouse. Similarly to situation observed in H-2^b/smouse, lack of co-localization, as well as areas of co-localized immunostaining may be found in relapsing H-2^bmouse. Note that NF160 immunostaining can not be well appreciated in merged image because of the bright MBP staining, although it is apparent when shown separately

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